Introduction: In winter, people in many areas have begun to choose to burn fires to warm them. The fire brings warmth to everyone, but it also brings hidden dangers: carbon monoxide poisoning. In winter, especially in the northern cities, there is a large number of carbon monoxide poisoning incidents every day, but we can avoid tragedies if proper precautions are taken and rescues are timely.
Why does carbon monoxide poisoning appear?
The binding force between carbon monoxide and hemoglobin is more than 200 times that of oxygen
Carbon monoxide binds very easily to hemoglobin in the blood. It binds hemoglobin more than 200 times as much as oxygen, and his presence prevents hemoglobin from binding to oxygen very well. When oxygen and hemoglobin were originally combined, hemoglobin would transfer oxygen to all parts of the body. However, because of the presence of carbon monoxide, oxygen in the body would be harmful to our lives.
How to deal with carbon monoxide poisoning?
Let oxygen race against time!
1, how to prevent?
In winter, whether it is a bath or an indoor activity, doors and windows cannot be closed. Even if only a small gap is left, this small gap may become a hero to prevent accidents and save lives!
2, how to deal with poisoning?
(1) Remove the poisoned person from the scene and place it in an air circulation environment
(2) Open the door and open the window for ventilation
(3) If you go to the hospital in time, you should not neglect treatment because you wake up and think it is okay.
Carbon monoxide poisoning has a condition called "delayed encephalopathy", which is the most serious complication of acute CO poisoning. In other words, some patients with CO poisoning experience a normal or nearly normal lapse after several days or weeks after the symptoms of acute poisoning have disappeared, and suddenly a group of neuropsychiatric symptoms, mainly dementia, reoccurs. This is clinically known as DNS. The clinical manifestations are very diverse, and may be mental and consciousness disorders, pyramidal and extrapyramidal dysfunction, or even partial dysfunction of the cerebral cortex, etc. The incidence of the disease is reported to be in the range of 0.2% to 47.3%, and in severe cases to 10%. % ~ 40%; It has also been reported that the incidence of delayed encephalopathy is as high as 25% to 50% in patients with conscious loss or acute CO poisoning with blood HbCO > 25%. A long period of coma has been considered as the DNS An important condition that occurs is: positive neurological signs, cardiovascular insufficiency, severe acidosis, abnormal brain CT or MRI examinations, and blood HbCO >25. Once diagnosed, patency should be promptly given hyperbaric oxygen therapy. "Keep high vigilance!
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